Q.What does the abbreviation IUD stand for?

Hormonal contraceptive pills (taken by females) work by blocking ovulation, altering cervical mucus and the endometrium -- they cannot act on spermatogenesis, which occurs in a different individual's body altogether.

Oral contraceptive pills contain synthetic oestrogen and/or progestogen and are taken by women to prevent pregnancy. Their genuine mechanisms of action include: inhibiting ovulation by suppressing the mid-cycle LH surge needed to trigger release of the egg; altering the consistency of cervical mucus so that it becomes thick and hostile to sperm transport; and modifying the uterine endometrium so that, even if fertilisation somehow occurred, the lining is unsuitable for implantation of the blastocyst. Since these pills act entirely within the female reproductive tract and endocrine system, they have no physiological pathway to inhibit spermatogenesis, which is the production of sperm inside the seminiferous tubules of the male testis -- an organ and process the pill never comes into contact with. This makes 'Inhibition of spermatogenesis' the incorrect action among the options.

Tubectomy is a terminal (permanent) method of birth control that surgically blocks the fallopian tubes.

Birth control methods are broadly natural, barrier, IUD/hormonal, and terminal (sterilisation) methods.

• Withdrawal method — a natural/behavioural method (coitus interruptus), not permanent.

• Implantation — not a contraceptive method at all; it refers to the attachment of the blastocyst to the uterine wall during pregnancy.

• Lactational amenorrhoea — a natural, temporary method relying on the absence of menstruation during intense breastfeeding.

• Tubectomy — a surgical sterilisation procedure in the female in which a small part of each fallopian tube is cut/tied/blocked, permanently preventing the ovum from meeting the sperm. Because it is permanent and irreversible in practice, it is classified as a terminal method.

Copper-T is a copper-releasing intra-uterine device (IUD); the copper ions it releases suppress sperm motility and thereby reduce the sperm's ability to fertilise the ovum.

Copper-T (Cu-T) is one of the most widely used intra-uterine devices (IUDs) for contraception. It works mainly through a chemical (not purely mechanical) mode of action:

• The copper wire wound around the device continuously releases Cu²⁺ ions into the uterine cavity.
• These copper ions create a spermicidal (toxic) environment in the uterus that suppresses sperm motility and reduces the sperm's capacity to fertilise the egg.
• Cu-T also makes the uterus less receptive for implantation, but the specific, textbook-defined primary mechanism asked here is the effect on sperm.

This distinguishes it from hormone-releasing IUDs (like LNG-20), which act mainly by making the cervix hostile to sperm and suppressing ovulation.

Oral contraceptive pills prevent pregnancy mainly by suppressing FSH and LH secretion, which blocks ovulation.

Oral contraceptive pills contain synthetic progestogens (often combined with estrogens). Through a negative-feedback effect on the hypothalamus and anterior pituitary, these hormones inhibit the secretion of GnRH and, in turn, FSH and LH, particularly preventing the mid-cycle LH surge that normally triggers ovulation -- so no egg is released and pregnancy cannot occur. Option (a), stimulating release of FSH and LH, is the opposite of what happens. Option (b), blocking the fallopian tube, describes tubal ligation/sterilisation, not the pill. Option (c), causing immediate degeneration of the released ovum, is not the pill's mechanism (it acts before ovulation, not after). The correct mechanism is suppression of the FSH/LH surge needed for ovulation.